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- We report a case of an acute ST-segment elevation myocardial infarction in a affected person with traditional cardiac danger factors using supraphysiologic doses of supplemental, intramuscular testosterone.
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- Direct myocardial cell damage may occur by disruption of mitochondria and induction of intrafibrillar collagen dysplasia (Melchert and Welder 1995; Sullivan et al 1998).
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We report a case of an acute ST-segment elevation myocardial infarction in a affected person with conventional cardiac risk factors using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person also had polycythemia, doubtless secondary to high-dose testosterone. The affected person underwent successful percutaneous intervention of the best coronary artery. Physicians and patients ought to pay consideration to the scientific penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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Erythrocytosis has been reported as a possible impact of testosterone, which may be useful in sufferers with anemia. The best risk of erythrocytosis happens with injectable types of testosterone compared with transdermal preparations, particularly in supraphysiologic doses. An increase of as a lot as 44% of the baseline hematocrit has been reported .
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and recommendations for monitoring. Testosterone replacement in hypogonadal males with angina improves ischaemic threshold and quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone improve human platelet thromboxane A2 receptor density and aggregation responses.
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